Mauricio Gil-Silva Lab Technician
- Characterization of supraspinal descending pathways that drive latent sensitization.
- BSA Neurobiology & Music Technology, Carnegie Mellon University
Currently pursuing a Masters in Epidemeology at Univerisity of Pittsburgh
Diogo Francisco da Silva dos Santos, PhD
- Characterization of supraspinal descending pathways that drive latent sensitization.
- MORCA analgesia and dependence
- Chronic pain of type II diabetes
Research Assistant, Aging Institute-University of Pittsburgh
Andy Cooper, PhD Postdoctoral Fellow
- Characterization of supraspinal descending pathways that drive latent sensitization.
- Investigation of the role of NPY in primary afferent fibers following nerve injury.
- PhD in Physiology and Pharmacology, University of Nottingham, UK
- BMedSc in Pharmacology, University of Birmingham, UK
Research Assistant, Center for Neuroscience, University of Glasgow
Andrew.Cooper.2@glasgow.ac.uk
https://www.gla.ac.uk/researchinstitutes/neurosciencepsychology/staff/andrewcooper/
Naomi Hedden
Lab Manager
- Characterization of supraspinal descending pathways involved in latent sensitization.
- BSc in Physiotherapy at University of Birmingham, UK
Peter Jukkola,
Lab Manager
- MORCA analgesia and dependence
PhD in Biomedical Sciences, Ohio State University
BS in Biology, Geneva College, Beaver Falls, PA
Lindsay Selan
Undergraduate
- Investigation of brain regions and neuronal populations involved in latent sensitization by examining Fos expression.
- Currently pursuing a bachelors in biology at University of Pittsburgh
Currently Pursuing a Masters in Cardiovascular Perfusion at Midwestern University
Tyler Nelson, PhD
-
Characterizing the neuropeptide Y1 receptor-expressing interneuron population in the spinal cord dorsal horn and its role in mediating neuropathic pain.
- BA/BS, Wofford College
- PhD, University of Pittsburgh
Post Doc, NYU
Ghanshyam Sinha
Assistant Research Professor
- How do spinal dorsal horn Y1R expressing neurons contribute to nerve injury associated pain? Do Y1 neurons' spinal circuitry transmit increased activity to supraspinal regions of the nervous system after injury? Is the increased activity long lasting?